Describe the classification.
Type I: sliding hiatal hernia
Most common type (95% of cases)
The GEJ and the gastric cardia slide up into the posterior mediastinum.
The gastric fundus remains below the diaphragm (hourglass stomach)
Type II: paraesophageal hiatal hernia
Part of the gastric fundus herniates into the thorax.
The GEJ remains in its anatomical position below the diaphragm.
Type III: mixed hiatal hernia
Mix of types I and II
The GEJ and a portion of the gastric fundus prolapse through the hiatus.
Type IV: complex hiatal hernia
Herniation of any abdominal structure other than the stomach (e.g., spleen, omentum, or colon)
Rarest type
Describe the anatomy for the pathophysiological understanding.
Esophageal hiatus
Central opening of the diaphragm, which allows the esophagus to pass through into the peritoneal cavity; forms the upper part of the esophageal sphincter and the reflux barrier
Formed by:
Left and right paravertebral tendinous crura
Median arcuate ligament
Gastroesophageal junction (GEJ)
Normally lies at the level of the esophageal hiatus
Phrenoesophageal ligament (PEL) attaches to the esophagus at the GEJ
Peritoneal fold that encircles the distal portion of the esophagus and gastroesophageal junction and connects them to the peritoneal surface of the diaphragm
Closes the esophageal hiatus and helps maintain the intra-abdominal position of the GEJ
Describe changes in the presence of a hiatal hernia.
Predisposing factors lead to laxity of the esophageal hiatus, e.g.:
Advanced age → phrenoesophageal ligament weakens
Smoking → loss of elastin fibres in the diaphragmatic crura
Obesity → deposition of fat in and around the crura → widened hiatus
Relative negative intrathoracic pressure and the lax hiatus → herniation of the abdominal contents into the thorax → loss of reflux barrier + compromised fluid emptying of distal esophagus → gastroesophageal reflux disease (GERD)
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