LEUKOPLAKIA
WHITE PLAQUE ON MUCOSA
PREMALIGNANT
HPV
SMOKELESS TOBACCO
DIFF
ORAL HAIRY LEUKOPLAKIA ( EXCLUSIVE TO HIV)
MALIGNANT MELANOMA
SQUAMOUS CELL CARCINOMA
hordoleon
stye
treatment and diagnosis
photo simple hordoleon
hordoleon definition\\CAUSES
ACUTE PLUGGING OF MEIBOMIAN GLAND AND INFLAMMATION RESULTS IN TENDER RED BUMP
CAUSES
BACTERIA IN EYE MAKEUP
ROSACEA SUFFERERS
SEBORRHEIC DERMATOSES SUFFERERS
HORDOLEON
ABSCESS FORMING
VS CHALAZION
NODULAR LESION ON EYELID NOT INFLAMED
HORDOLEON PE
APPEARS IN A DAY
EXTERNAL ON EYELASH FOLLICLE
OR INTERNAL INFLAMMATION MEIBOMIAN GLAND -SWELLING UNDER CONJUNCTIVE
STAPH AUREUS MAIN CAUSE
HORDOLEON TREATMENT
RESOLVES SPONTANEOUSLY OVER 1—2 WEEKS
LITTLE DATA ON TREATMENT - EXPERT CONSENSUS
FACILITATE DRAINAGE -MOIST WARM COMPRESSES
4 X DAY FOR 5-10 MINUTES
MASSAGE AND GENTLE WIPING OF EYE AFTER MOIST CMPRESS( WITH CLEAN HANDS)
NO EYE MAKE UP
NO IMPROVEMENT TWO WEEKS -REFER OPTHALMOLOGIST
PT WITH FREQUENT HORDOLEUM MAY RESPOND TO TOPICAL ANTIBIOTIC /CORTICOSTEROID OINTMENT COMBO- BUT THESE PATIENT MUST BE MANAGED BY OPTHALM - * RISKS WITH TOPICAL GLUCOCORTISOID USE EYE
HORDOLEUM
UNCOMMON COMPLICATION
PRE SEPTAL CELLULITIS
HORDOLEUM PRE SEPTAL CELLULITIS MGT
OUTPATIENT ADULT MILD PRESEPTAL CELLULITIS CAN USE ABX AND CLOSE FOLLOW UP
ABX AUGMENTIN ( FOR STAPH AUREUS) MRSA IS NOT COMMON IN THIS SETTING
(PCN ALLERGY USE SND OR THIRD GEN CEPHALOSPORIN EG CEFDINIR, CEFPOXIDINE, CEFUROXIME)
IF NOT IMPROVEMENT IN 24-48 HOURS COVER FOR MRSA-B
REMEMBER MRSA B- BACTRIM DS 160/800 Q 12 HOURLY
PLUS
ACTIVE AGAINST GASTREP AND H INFLUENZAE -
AUGMENTIN Q 12
OR CEPHALOSPORIN EG CEFDINIR, CEFPOXIDINE, CEFUROXIME)
SHOULD RESPOND RAPIDLY
MONO
TETRAD OF SYMPTOMS
FATIGUE
FEVER
LYMPHADENOPATHY( POSTERIOR CERVICAL NODES)
PHARYNGITIS ( 10% HAVE STREP)
ETIOLOGIC AGENT OF MONO IS THE EPSTEIN BARR VIRUS
AVOID AMOXICILLIN, CAN CAUSE RASH -NOT ALLERGY BUT EBV REACTING WITH PCN MOLECULE
HORDOLEUM DIFF DX
CHALAZION
MOLLUSCUM CONTAGIOSUM ON EYELID
DIFF DX
PRE SEPTAL CELLULITIS-USU MILD
VS ORBITAL CELLULITIS -SERIOUS CAN CAUSE LOSS OF VISION
ORBITAL USU HAVE PAIN WIH EYE MOVENENTS, PROPTOSIS-PROTRUSION OF EYEBALL, OPTHALMOPLEGIA-PARALYSIS OR WEAKNESS OF EYE MUSCLES- CAN AFFECT III,IV OR VI,BLURRY VISION, DIPLOPIA, PTOSIS , DYSPHAGIA!
PRESBYCUSIS
CARDINAL SIGNS
CANNOT HEAR CONSONANTS
SYMMETRICAL
CANNOT HEAR SPEECH IN CROWDED ROOM
LOSS OF HIGH PITCHED SOUNDS
CONJUNCTIVITIS ETIOLOGY RISK FACTORS
70% VIRAL
15% ALLERGIC
POOR HAND WASHING
HIGHLY CONTAGIOUS
SPREAD IN INSTITUTIONAL SETTINGS
ADENOVIRUS OR COMMON COLD COMMONEST CAUSE
EYEDROP MEDICATIONS CAN CAUSE-VASODILATORS
PAPILLEDEMA
ETIOLOGY
SWELLING OF OPTIC NERVE HEAD AND DISC 2/2 >ICP
CARDINAL SYMPTOM OF ICP IS HEADACHE, PAPILLEDEMA IS 2NDRY
VIRAL CONJUNCTTIVITIS
HSV
RUBELLA
EPSTEIN BARR ( MAIN MONO)
MOLLUSCUM CONTAGIOSUM (POX) SUSPECT HIV IF IN ADULT
VZV
LASTS 5-10 DAYS
CONTAGIOUS IF TEARING 1 WEEK
COXSACKIE
CONJUNCTIVITIS
PE
RED EYE INJECTION
WATERY DISCHARGE
RECENT RESPIRATORY INFECTION IF ADENOVIRUS
BURNING
ITCHING
PHOTOPHOBIA
VISION NOT AFFECTED
FOREIGN BODY SENSATION
RESOLVES 1-2 WEEKS
CONJUNCTIVITIS TREATMENT
ANTIHISTAMINE /DECONGESTANT EYE DROPS-
OLAPATADINE =PATADAY OR PATANOL
VISINE AC
ARTIFICIAL TEARS
COOL COMPRESS
REFER OPTHALMOLOGIST IF >10 DAYS OR CORNEAL INVOLVEMENT
FOLLICLE PAPILLAE
VIRAL CONJUNCTIVITIS
GONORHHEAL CONJUNCTIVITIS
HYPERACUTE
PURULENT COPIOUS DRAINAGE ,
EYES GLUED SHUT
REFER TO OPTHALMOLOGIST
CORNEAL INVOLVED < 2 DAYS
PERMANENT VISION LOSS
CHLAMYDIAL CONJUNCTIVITS PRESENTS WITH GENITAL LESIONS TOO
RED FLAGS
Diminished visual acuity
Photophobia
Severe foreign body sensation preventing patient from keeping eye open
Corneal opacity
Fixed pupil
Severe headache with nausea (angle-closure glaucoma)
Proptosis
LACK OF RESPONSE TO THERAPY
CANNOT OPEN EYE
DIFF DIAGNOSIS
Foreign body
Uveitis, iritis, scleritis UVEA IS SCLERA, IRIS AND CHOROID( BLOOD VESSELS)
Corneal abrasion
Dacryocystitis INFLAMMATION OFLACRIMAL SAC
Hyphema IS WHEN
BLOOD COLLECTS IN FRONT OF EYE, BETWEEN CORNEA AND IRIS
Angle-closure glaucoma
Subconjunctival hemorrhage
Periorbital cellulitis
BACTERIAL CONJUNCTIVITIS
ADULTS
STAPH AUREUS
CHILDREN, HEMOPHILUS INFLUENZAE
STREPTOCOCCUS PNEUMONIAE
bACTERIAL CONJUNCTIVITIS
PHARM MGT
PCP NEVER PRESCRIBE TOPICAL GLUCOCORTISOIDS-MAY THREATEN VISION
AZITHROMYCIN MACROLIDE 1DRP BID THEN DAILY
AMINOGLYCOSIDES :
GENTAMYCIN 1-2 DROPS 0.3 %.OINTMENT FORM ALSO
TOBRAMYCIN OINTMENT OR DROPS 0.3%
FLUOROQUINOLONES: FIRST LINE FOR CONTACT USERS
LEVOFLOXACIN 0.5% DROPS
CIPROFLOXACIN 0.3% DROPS
OFLOXACIN 0.3%
ACUTE ALLERGIC CONUNCTIVITIS
symptoms
RHINITIS IN 50%
HEADACHE
POSITIVE FAMILY HISTORY ASTHMA HAYFEVER ATOPY
BOTH EYES
CLEAR WHITE STRINGY ROPY DISCHARGE
PAINLESS
ALLERGIC SHINERS
COBBLESTONE UPPER INNER EYELIDS
COBBLESTONE OF ALLERGIC
ALLERGIC CONOJUNCTIVITIS
TREATMENT
IDENTIFY ALLERGEN AND REMOVE IT
CLARITIN-LORATIDINE 2ND GEN ANTIHISTAMINE
ALLEGRA- FEXOFENADINE-2ND GEN AH
BENADRYL
REMOVE CONTACTS
ANTIHISTAMINE VASOCONSTRICTOR( PATANOL0 DROPS CAN CAUSE REBOUND VASODILATION IF USED >2 WEEKS
EDUCATION AND PREVENTION
HAND WASHING
THROW OUT OLD LENS SOLUTION , LENSES, HOLDERS
WASH BEDDING
NO POOLS
GLAUCOMA
TYPES
DEFINITION
OPEN ANGLE
CHRONIC OPEN ANGLE
ANGLE CLOSURE
ALLT YPES TEH NERVE CONNECTING THE EYE TO THE BRAIN IS DAMAGED , USU 2/2 HIGH EYE PRESSUE
POAG
PRIMARY OPEN ANGLE GLAUCOMA
RISK FACTORS
BLACK PEOPLE, LEADING CAUSE OF BLINDNESS
STEROID USE
ELDERLY
HTN
DM
FAMILY HISTORY
SLOW VISION LOSS, MOST COMMON
ASST FINDINGS
USU ASYMPTOMATIC SLOW VISION LOSS, MOST COMMON
>IOP INTRAOCULAR PRESSURE
>22MHG
HALOS
NOTCH IN CUP
CUPPING- REFER TO OPTHALMOLOGIST
DECREASED PERIPHERAL THEN CENTRAL VISION
ACUTE ANGLE CLOSURE
CLOSED ANGLE , NARROW ANGLE
SUDDEN INCREASE IN IOP
SUDDEN VISION LOSS
RAPID PERIPHERAL LOSS THEN CENTRAL
FIXED AND DILATED PUPIL THAT LOOKS OVAL
SUDDEN HEADACHE NAUSEA VOMIITING
ER REFER
ASIANS AND ESKIMOS
RARE
ACUTE CLOSED ANGLE GLAUCOMA
GLAUCOMA DIFF DX
UVEITIS
MACULAR DEGENERATION
FOREIGN BODY
NON PHARM SX
SURGERY OR LASER TX TO REDUCE IOP
PHARM:
TOPICAL BETA ADRENERGIC BLOCKERS
LEVOBUNOLOL TIMOLOL
MIOTICS: DRUGS THAT CAUSE THE PUPIL TO CONSTRICT, ALLOWING BLOCKED DRAINAGE ANGLE TO OPEN
PILOCARPINE GLAUCOMA ONLY
SYSTEMIC AGENTS??
BRINZOLAMIDE ( AZOPT) I1 DROP
MEDS AND GLAUCOMA
DO NOT PRESCRIBE BB EYE DROPS TO SOMEONE ON THEM ORALLY
MEDS THAT CAN CAUSE GLAUCOMA:
STEROIDS
DECONGESTANT
ANTHISTAMINES-DRAMAMINE-DIMENHYDRAMINE. 1ST GEN AH
PRESCR MEDS FOR BLADDER INCONTINENCE
DIET PILLS?? TOPIRAMATE- CONTAINS SULFA
SULFA
ALWAYS CHECK W YOUR OPTHALMOLOGIST BEFORE NEW MEDS IF YOU HAVE GLAUCOMA
MACULAR DEGENRATION
MOST COMMON CAUSE OF VISION LOSS IN ELDERLY
SMOKERS
ATROPHIC OR DRY FORM -MORE COMMON LESS SEVERE
WET FORM - BLINDNESS 80% VISION LOSS IN PEOPLE
IST SIGN SCOTOMA-BLIND SPOT
PERIPHERAL AND COLOR VISION NORMAL
REFER
AREDS
DC SMOKING
EAR DISORDERS
ANATOMY
OTITIS EXTERNA
FACTS AND BACTERIA
Approximately 98% of acute otitis externa cases in North America are bacterial.
SWIMMERS EAR
INVOLVES EXTERNAL AUDITORY CANAL AURICLE OR BOTH
Bacterial
Fungal
Pseudomonas (most common pathogen)
Staphylococcus
Streptococcus
Aspergillus (most common fungal pathogen)
Candida albicans
OTOTOXIC DRUGS
ASPIRIN
LOOP DIURETICS
ABX: AMINOGLYCOSIDES
VANCOMYCIN
ERYTHROMYCIN
SILDAFENIL
CISPLATIN
ANTIMALRIA DRUGS
Tragal PALPATION and/or pinna pain
Traction of the pinna elicits pain: hallmark sign of OE
Otalgia/conductive hearing loss
Edema and redness in the external auditory canal
Itching in the external auditory canal
Otorrhea(EAR DRAINAGE)/purulent discharge in external canal; usually foul smelling
Discharge characteristics
Acute bacterial: scant white mucus; may be thick
Chronic bacterial: bloody if granulation tissue present
Fungal: fluffy and white to off-white; may be black, gray, bluish-green, or yellow
Normal tympanic membrane (TM)
Fever: occasional
Tinnitus
Bilateral involvement: rare
EAC pain while chewing
Regional lymphadenitis may be present
OTITIS EXTERNA TREATMENT
CIPROFLOXACIN AND HYDROCORTISON -CIPRO HC OTIC
3 DROPS BID X 7 DAYS
*DONT GIVE IF PERFORATED TYMPANIC MEMBRANE OR TYMPANOSTOMYTUBES
CIPROFLOXACIN /DEXAMETHASONE OTIC 4 DROPS BID 7 DAYS
OXOFLOXACIN
CAN BE USED WITH T TUBES
OTITIS MEDIA ACUTE
SIGNS
MIDDLE EAR EFFUSION
*BULGING TYMPANIC MEMBRANE,
*REDUCED MOBILITY OF TYMPANIC MEMBRANE-PNEUMATIC PRESSURE
*INTENSE ERRYTHEMA OF TM
DISTORTED LANDMARKS TM
OPAQUE TM ( FLUID-CAN BE CLOUDY OR YELLOW )
TRANSIENT HEARING LOSS
aom
DIAGNOSTIC TESTING
HANDHELD OTOSCOPE
*STANDARD FOR DX AOM PNEUMATIC OTOSCOPE FOR TM MOBILITY
TYMPANOMETRY
AOM DIFF DIAGNOSES
OTITIS MEDIA WITH EFFUSION (OME):
NO PAIN/MILD PAIN
FULL FEELING
DIMINISHED HEARING
NO BULGING TM
CHOLESTEATOMA
TMD
DENTAL CAUSES
AOM
NON PHARM
PHARM
DIAGNOSTICS
NON
LOCAL HEAT
MYRINGOTOMY ( TUBES)
NO DIAGNOSTICS INDICATED EXCEPT CULTURE IF REPEATS
IBUPROFEN TYLENOL
*NO DECONGESTANTS /ANTIHISTAMINES-PROLONG MEE
ABX AUGMENTIN 500 TID
PCN ALLERGY
CEFURIXIME OR CEFUROXIME( 2ND GEN CEPH)
AZITHROMYCIN MACROLIDE.500 MG X 5 DAYS
*CIPRO/DEXAMETH OTIC -FOR PT WITH TUBES ONLY
AOM FOLLOW UP AND RED FLAGS
FLAGS
IF NO IMPROVEMENT 48-72 HOURS CONSIDER SWITCHING TO 2ND LINE
ENT referral for recurrent OM (three presentations in 6 months or four in 1 year)
Emergency treatment needed for signs of mastoiditis and/or meningitis
F/U CALL IN 72 HOURS
UPPER RESPIRATORY
HEENT NODES
ALLERGIC RHINITIS
ETIOLOGY AND RISK
DEFINITIVE DX REQUIRES SPECIFIC IGE REACTIVITY ( ALLERGIST REF)
20-30% ADOLESCENTS
SEASONAL
PERENNIAL
LESSENS WITH AGE
ASSESSMENT FINDINGS
ALLERGIC SALUTE
DRY MOUTH/MOUTH BREATHING
COBBLESTONE PHARYNX AND TONSILS
LYMPH NODES
CONJUNCTIVAL INJECTION
PALE BOGGY TURBINATES
CLEAR NASAL SECRETIONS
rupture of mast cells and release of histamines, leukotrienes, prostaglandins,
INTRANASAL STEROIDS (INS)
FLONASE 1 SPRAY EACH NOSTRIL DAILY
TRIAMCINOLONE NASACORT 2 DAILY
BECLOMETHASONE 1 SPRAY DAILY
ST GEN ANTIHISTAMINE:
BENADRYL/ HYDROXYZINE-ATARAX
2ND GEN A/H LESS DROWSY
CETIRIZINE( ZYRTEC
LEVOCETRIZINE XYZAL
LORATADINE CLARITIN
FEXOFENADINE, ALLEGRA
ASTELAZINE 2 SPRAYS DAILY
PATANASE
(2ND TREATMENT INS- AHS
AHS-INS
ORAL AHS-ORAL DECONG)
CAN ADD SINGULAIR MMONOLEUKOTRIENE RECEPTOR ANTAGONIST
DECONGESTANTS CAUSE REBOUND DECONGESTION DUE TO WEAKENED AFFINITY FOR ALPHA ADRENERGIC RECEPTOR
DIAGNOSTIC TESTING AND FOLLOW UP
NO IMAGING,
REFER TO ALLERGIST
SURGICAL REDUCTION OF TURBINATES
IGE ALLERGY TESTING SKIN OR BLOOD FOR PT WHO DOES NOT RESPOND TO EMPIRIC TREATMENT
OR REFER TO SOMEONE WHO TESTS
PHARY/TONSI
VIRAL
ADENOVIRUS
RHINOVIRUS
RSV
HIV
BACTERIAL :
GABHS
HEMOPHILUS INFLUENZAE
MYCOPLASMA PNEUMONIAE ATYPICAL
CHLAMYDIA/ GONORRHEA
PHARYNGITIS/TONSILITIS
SORE THROAT
PHARYNGEAL EDEMA
TONSILLAR EXUDATE +/OR ENLARGED TONSILS
MALAISE
ACCURATE FINDINGS NEEDED FOR STREP - PREVENT GABHS GLOMERULONEPHRITIS
PERITONSILLAR ABSCESS
MASTOIDITIS
GABHS INFECTION CENTOR CRITERIA
3-4 POINTS TREAT EMPIRICALLY FOR STREP
2 POINTS : RADT + TX IF POS+
1 POINT : STREP UNLIKELY
0-1 POINT: NO STREP NEEDED
RADT IS RAPID ANTIGEN DETECTION TEST-STREP TEST
PHARYN
URI
TONSILITIS
EPIGLOTTITIS
PHARYNG/TONIS
DIAGNOSTIC STUDIES
RABT 95-99% SPECIFIC
SWAB FROM TONSILS + PHARYNGEAL WALL
NO STREP TEST IF VIRAL SYMPTOMS
(RHINORRHEA ORAL ULCERS COUGH)
CBC FOR POLYPS
10% OF PTS WITH MONO HAVE STREP ALSO
ASO DETECTS PAST INFECTION -NOT PRESENT
PHARYN/TONIS
Gargling with warm salt water
Increased fluid intake
Patient education about disease, course and treatment
Change toothbrush after treatment
PHARYNG.TONS
TYLENOL /NSAID
PCN G X 1 DOSE IM
PEN VK X 10 DAYS
AMOXICILLIN X 10 DAYS
AZITHROMYCIN( NOT FIRST LINE, GOOD CHOICE FOR ALLERGIES)
CEPHALEXIN BID X 1O DAYS 1ST GEN CEPH
CLINDAMYCIN MACROLIDE
CLARITHROM( BIAXIN) MACROLIDE
Acute rhinosinusitis
INFLAMMATION NASAL CAVITY PARANASAL SINUSES
<4 WEEKS ACUE
4-12 WEEKS SUBACUTE
>12 WEEKS CHRONIC CRS
4OR > EPISODES A YEAR RECURRENT ARS (RARS)
SMOKERS SWIMMING BAD H2O
COMMON COLD '
SEPTUM DEVIATION
NASAL POLYPS
ARS
, CORONAVIRUS
FLU A B , PARA
BACTERIAL
STREP PNEUM -MOST
HEMOPHILUS INFLU -SMOKERS
MORAXELLA CATARHALIS
BACTERIAL CRS
STAPH +
PSEUDOMONAS -
PREGNANCY -PHYSIOLOGIC NASAL CONGESTION
SYMPTOMS
3 MAIN
PURULENT NASAL DISCHARGE
NASAL OBSTRUCTION
FACE PAIN PRESSURE FULLNESS
(FRONTAL FOREHEAD
MAXILLARY CHEEK TEETH
ETHMOID EYES)
BACTERIAL VS VIRAL GUIDELINES
MILD SYMPTOMS 1ST 3 DAYS
DONT WORSEN ,<10 DAYS
> 10 DAYS
SYMPTOMS WORSEN
HIGH FEVER >102
SEVERE SYMPTOMS 1ST 3 DAYS
DOUBLE SICKENING
pe
3 CARDINAL SIGNS
PURULENT DISCHARGE
FACE PAIN/PRESSURE/FULLNESS
NASAL TURBINATE EDEMA ERYTHEMA AND DISCHARGE
HYPONASAL SPEECH
REDUCED OR ABSENT SMELL
CHECK FOR NOSE SYMMETRY PATENCY OF NARES
RED THROAT ( PND)
EXAM EYES
PALPATE SINUSES
EXAM EARS (R/O OTITIS MEDIA)
TAP EXAMINE TEETH
TRANSILLUMINATION -NOT ALWAYS ACCURATE
rhinosinusitis and crs
diff diagnoses
Viral URI
Allergic rhinitis
Nonallergic rhinitis (triggered by strong odors or change in temperatures)
Dental abscess
Headaches
Nasal foreign body
Wegener’s granulomatosis
EMPIRIC PRESUMPTIVE DIAGNOSIS
CT IMAGING NOT APPROPRIATE FOR UNCOMPLICATED ARS. FOR SEVERE HEADACHE, FACILAL SWELLING, CRANIAL NERVE PALSIES
CT AFTER MEDICAL MGT,NEURO DEFICITS OR IMMUNOCOMPROMIISE
Avoid environmental irritants (e.g., cigarette smoke)
Manage allergic rhinitis appropriately
Humidified air can improve mucus clearance
Look for otitis media when evaluating patient with rhinosinusitis (and vice versa)
Increase fluid intake
Sleep with head of bed elevated to aid with drainage
EDUCATE ON NO ABX RIGHT AWAY
PHARM ALL
TYLENOL
NSAID
INS FLONASE 1 SPRAY BID
NEIL MED SALINE NASAL IRRIGATION
DECONGESTANTS/ANTIHISTAMINES NOT SHOWN TO BE EFFECTIVE FOR BACTERIAL SINUSITIS
ABX:
AMINOGLYCOSIDES( EG GENT)
SILDENAFIL
ANTIMALARIA MEDS
ARS BACTERIAL
ABX
AMOXIL 500 MG Q8 X5-10 DAYS OR
875 MG Q 12 X 5—10 DAYS
AUGMENTIN 500/125 Q 8 X 5-10 DAYS
875/125 Q 12 X 5-10 DAYS ,IF TRIED AMOXIL IN LAST MONTH
AUGMENTINXR 2000 /125 BID X 10 DAYS, RECENT ABX USE RECENT HOSPTALIZATION(5 DAYS) HIGH RESISTANCE, RX FAILURE, COMORB
LEVAQUIN 500 MG OD X 10-14 DAYS OR 750 OD X 5 DAYS
DOXY-TETRACYCLINE 100 BID X 10 DAYS
MOXIFLOXACIN (AVELOX) 400 MG OD X 10 DAYS
2ND LINE
CLINDAMYCIN
3RD GEN
CEFIXIME
CEFPODIXIME
REFERRAL
HIGH PERSISTENT FEVER F>102
PERIORBITAL EDEMA, ERYTHEMA
CRANIAL NERVE PALSIES
DIPLOPIA, PROPTOSIS ( EYE PROTRUSION), AOMS ABNORMAL
SEVERE HEADACHE
AMS
MENINGITIS SIGNS
chronic sinusitis
etiology
risk factors
often missed by clinicians
nasal obstruction
nasal polyps
facial congestion/pressure
discolored nasal discharge
hyposmia
halitosis
2 or more symptoms >12 weeks is crs
periorbital edema
1in 5 have asthma ,
allergies , smoke exposure, cystic fibrosis , ciliary dyskinesia
immunodeficiency
crs
SINUS ENDOSCOPY FIRST LINE
GOLD STANDARD TESTING IS CT SCAN-DONE FOR COMPLICATED OR SURGERY TO BE DONE
ALLERGY TESTING IGE if suspected cause
CRS
SALINE IRRIGATION
INS STEROIDS FOR 8 -12 WEEKS
FOLLOW UP CARE
F/U 3-5 DAYS AFTER INITIATION OF ABX THERAPY TO SEE IF IMPROVED
REFER TO ENT - RECURRENT INFECTIONS AFTER MAXIMAL MEDICAL THERAPY
ALLERGIST
ER IF MENINGITIS SUSPECTED
FINDINGS
LIPOGRNAULOMATOUS INFLAMMATION OF EYELID FROM INFLAMED AND OBSTRUCTED SEBACEOUS GLANDS OF EYELID
MOST COMMON EYELID LESION
RISK FACTORS:
STYE ( HORDEOLUM) CAN DEVELOP INTO ONE
EYELASH MITES
ROSACEA
TRAVEL TO TB COUNTRIES
NON TENDER PALPABLE NODULE FOR MONTHS, CAN BE INNER MARGIN
CORNEAL ASTYGMATISM can develop FROM PRESSURE ON CORNEA
DIFF DIAGNOSIS CHALAZION
STYE
MOLLUSCUM
LEISHMANIASIS
CELLULITIS
XANTHELASMA
PAPILLOMA
CHAALZION
FOLLOW UP
NONE, IF PAIN DO FLUORESCIN STAIN TO ASSESS CORNEAL DAMAGE
Chalazia are inflammatory, not infectious. Antibiotics only appropriate for associated infection. Majority of chalazia respond well to conservative management. Refer patients with recurrent or refractory chalazia-OPTHALMOLOGIST
WHO MAY DO BOTOX, EXCISION, STEROIDS
2-4 WEEK F/U IF SMALL AND SIMPLE
CHALAZION TREATMENT
NON PHARM AND PHARM
Warm, moist compresses on affected lid for 15 minutes 2-4 times per day
Baby shampoo application and eyelid massage
DOXYCYCLINE 100 BID X 10 DAYS OR MINOCYCLINE 50 X 10/7
METRONIDAZOLE IF TETRA CONTRA
REFER TO OPTHAL FOR INTRALESIONAL STEROIDS
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